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Emma Hartnell-Baker’s work shows that when children are screened for speech sound processing on entry and supported immediately, they do not go on to struggle with reading and spelling. In the schools she supports, this has eliminated the need for dyslexia assessment referrals. This challenges the idea that dyslexia is inevitable or solely biological, suggesting instead that the difficulties it describes can often be prevented when early needs are identified and addressed. Where children have already experienced difficulty, the label can still provide protection and access to support, but the focus should be on ensuring fewer children require that label in the first place.

Are children “born dyslexic”?

Were children “born dyslexic”? Are children born with dyslexia?
Or does what we call dyslexia develop when early needs aren’t identified and addressed?


Many people have been told that dyslexia is something a child is born with, in the same way as a lifelong condition such as ADHD. This belief is widespread, but it does not accurately reflect how dyslexia is identified or how literacy develops.

Dyslexia is not diagnosed at birth. It is identified when a child shows persistent difficulty with accurate and fluent reading and spelling, relative to age and opportunity (Lyon, Shaywitz, & Shaywitz, 2003; Peterson & Pennington, 2012).

This means that dyslexia is defined by observable literacy difficulty, not biology alone.

If a child does not struggle with reading and spelling, they are not identified as dyslexic within current diagnostic frameworks.


Biological Risk Does Not Mean Inevitable Difficulty


There is strong evidence that literacy difficulties are influenced by heritable and neurocognitive factors, particularly in relation to speech sound processing and language development (Pennington & Bishop, 2009; Snowling & Melby-Lervåg, 2016).

However, developmental models do not support a deterministic interpretation. The multiple deficit model demonstrates that outcomes arise from the interaction of risk and protective factors, rather than a single causal pathway (Pennington, 2006). Early language and phonological processing are consistently identified as key predictors of later literacy development (Snowling & Hulme, 2012).

Biological vulnerability therefore increases the likelihood of difficulty, but does not make literacy failure inevitable.


What Happens When Early Risk Is Not Identified


In many educational contexts, children are not routinely screened for differences in speech sound processing when they begin school. Instruction proceeds on the assumption that learners can readily map speech to print, despite evidence that this process varies significantly across children (Snowling & Hulme, 2012).

Where early differences are not identified, children are more likely to experience difficulty establishing accurate phoneme–grapheme correspondences, which underpin both reading and spelling (Ehri, 2005).

This may lead to:

  • difficulty mapping speech sounds to written forms (Ehri, 2005)

  • reliance on compensatory strategies such as guessing or memorisation (Share, 1995; Perfetti, 2007)

  • reduced accuracy in reading and spelling (Snowling & Melby-Lervåg, 2016)

Over time, these patterns can become established. It is typically at this stage that children are referred for assessment and may be identified as dyslexic (Peterson & Pennington, 2012).


Evidence for Early Prevention (preventing the need for Intervention)


There is strong evidence that early intervention targeting phonological and language-related skills improves literacy outcomes and reduces the severity of later reading difficulties (Ehri, 2005; Hulme & Snowling, 2013; Snowling & Hulme, 2021).

Such interventions support the development of phoneme awareness and its connection to print, which are central to word reading and spelling acquisition (Ehri, 2005).

However, most research evaluates intervention after difficulty has begun to emerge, rather than examining fully preventative models implemented at school entry.


A Prevention Model


Where children are systematically screened for speech sound processing differences on entry to school, and receive immediate, precise support, a different developmental trajectory may be observed.

In such contexts:

  • children acquire accurate and fluent reading and spelling

  • later intervention is not required

  • referrals for dyslexia assessment do not arise

This observation is consistent with evidence that early, targeted support reduces the likelihood of persistent literacy difficulty (Hulme & Snowling, 2013), while extending it by suggesting that the conditions under which a dyslexia diagnosis is made may not emerge when early risk is fully addressed.

This interpretation does not negate biological variation. It indicates that biological risk does not inevitably lead to literacy failure when appropriate instructional responses are in place (Pennington, 2006).


Diane McGuinness and the Role of Instruction


Diane McGuinness argued that many reading difficulties attributed to dyslexia can be explained by insufficiently explicit instruction in the alphabetic principle and phoneme–grapheme correspondences (McGuinness, 2004, 2005).

Her work emphasised that when the structure of the writing system is made clear and consistent, a far greater proportion of learners achieve successful reading outcomes.

This aligns with evidence demonstrating the importance of explicit instruction in supporting the development of accurate word reading and spelling (Ehri, 2005).


Julian Elliott, the Delphi Definition, and the Role of the Label


Julian Elliott has argued that dyslexia is not a clearly distinguishable condition, but a label applied to individuals who experience reading difficulty (Elliott & Grigorenko, 2014). There is no clear boundary separating “dyslexic” readers from other poor readers, and the label itself does not determine the nature of effective instruction.


This position aligns with the international Delphi consensus, which defines dyslexia as a persistent difficulty in accurate and fluent word reading and spelling, typically associated with weaknesses in phonological processing, and occurring despite appropriate instruction (Snowling et al., 2020).


Taken together, these perspectives clarify that dyslexia:

  • is a description of literacy difficulty, not a distinct type of thinker

  • is not identified independently of reading and spelling performance

  • is not defined by strengths or a particular cognitive style


While some individuals who have experienced literacy difficulty go on to demonstrate resilience or creativity, these are best understood as responses to challenge, not inherent features of dyslexia (Elliott & Grigorenko, 2014).


Instructional Casualty and Why Diagnosis Still Matters


Where children experience persistent literacy difficulty, it is appropriate to consider them as instructional casualties, reflecting a mismatch between their learning needs and the instruction they received.

In these cases, the dyslexia label can serve an important function.

Although it does not change the nature of instruction required, it may:

  • provide a clear explanation that reduces self-blame

  • support access arrangements and reasonable adjustments

  • offer recognition and protection within educational systems


Diagnosis should not be withheld from learners who have already experienced difficulty.

In this context, the label operates as a protective mechanism, not as a determinant of how a child learns.


Rethinking the Narrative


The increasing tendency to describe dyslexia as a distinct way of thinking, often associated with strengths, is not supported by the evidence base.

A more accurate interpretation is that:

  • some learners face early challenges in mapping speech to print

  • some go on to develop strengths in response to those challenges

  • those strengths are shaped by experience, not caused by dyslexia itself

From this perspective, individuals may succeed despite early literacy difficulty, not because of it.


A Shift in Focus


The central question is not:

Was the child born dyslexic?

But rather:

Was the child’s early risk identified and supported in time?

Where early needs are met, persistent literacy difficulty may not emerge. Where they are not, difficulty becomes visible and is subsequently labelled.


Conclusion


Dyslexia is identified through observable difficulty in reading and spelling, not through biology alone.

Biological differences contribute to risk, but they do not determine outcome in isolation.

Evidence from developmental and instructional research, alongside the work of McGuinness (2004, 2005), Elliott and Grigorenko (2014), and the Delphi consensus (Snowling et al., 2020), supports a more precise interpretation:

  • some children are more vulnerable to difficulty

  • that vulnerability is real

  • but the outcome depends on early identification and instructional response

When early risk is identified and addressed effectively, the literacy difficulties associated with dyslexia may be substantially reduced or may not emerge.


References

Ehri, L. C. (2005). Learning to read words: Theory, findings, and issues. Scientific Studies of Reading, 9(2), 167–188.

Elliott, J. G., & Grigorenko, E. L. (2014). The Dyslexia Debate. Cambridge University Press.

Hulme, C., & Snowling, M. J. (2013). Learning to read: What we know and what we need to understand better. Child Development Perspectives, 7(1), 1–5.

Lyon, G. R., Shaywitz, S. E., & Shaywitz, B. A. (2003). A definition of dyslexia. Annals of Dyslexia, 53, 1–14.

McGuinness, D. (2004). Early reading instruction: What science really tells us about how to teach reading. MIT Press.

McGuinness, D. (2005). Language development and learning to read. MIT Press.

Pennington, B. F. (2006). From single to multiple deficit models of developmental disorders. Cognition, 101(2), 385–413.

Pennington, B. F., & Bishop, D. V. M. (2009). Relations among speech, language, and reading disorders. Annual Review of Psychology, 60, 283–306.

Perfetti, C. A. (2007). Reading ability: Lexical quality to comprehension. Scientific Studies of Reading, 11(4), 357–383.

Peterson, R. L., & Pennington, B. F. (2012). Developmental dyslexia. The Lancet, 379(9830), 1997–2007.

Share, D. L. (1995). Phonological recoding and self-teaching. Cognition, 55(2), 151–218.

Snowling, M. J., & Hulme, C. (2012). Annual research review. Journal of Child Psychology and Psychiatry, 53(5), 593–617.

Snowling, M. J., & Hulme, C. (2021). Reading disorders and dyslexia. Current Opinion in Pediatrics, 33(6), 731–735.

Snowling, M. J., Hulme, C., Nation, K., & others. (2020). Dyslexia: A consensus statement. Journal of Child Psychology and Psychiatry.

Snowling, M. J., & Melby-Lervåg, M. (2016). Oral language deficits in familial dyslexia. Psychological Bulletin, 142(5), 498–545.

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